Polioencephalomalacia
http://dbpedia.org/resource/Polioencephalomalacia an entity of type: Thing
Die Zerebrokortikalnekrose (Cerebrocorticalnekrose, CCN, „Hirnrindennekrose“) ist eine bei Wiederkäuern auftretende metabolisch-toxische Gehirnerkrankung (Enzephalopathie) infolge eines Mangels an Thiamin (Vitamin B1). Sie entspricht damit der Beriberi bzw. der Wernicke-Enzephalopathie des Menschen, der Chastek-Paralyse der Pelztiere und der Thiaminmangel-Enzephalopathie der Katze.
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Polioencephalomalacia (PEM), also referred to as cerebrocortical necrosis (CCN), is a neurological disease seen in ruminants that is caused by multiple factors, one of which is thiamine depletion in the body. Thiamine (vitamin B1) is a key chemical in glucose metabolism that, when deficient, is most threatening to neurological activity. In addition to altered thiamine status, an association with high sulfur intake has been observed as a potential cause of PEM. Other toxic or metabolic diseases (eg, acute lead poisoning, sodium toxicosis/water deprivation) can result in PEM as well. Cattle, sheep, goat, and other ruminants that are diagnosed with PEM or pre-PEM suffer opisthotonus, cortical blindness, disoriented movement, and eventually fatality, if left untreated. Current data shows that
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Zerebrokortikalnekrose
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Polioencephalomalacia
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4521853
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1101482075
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Die Zerebrokortikalnekrose (Cerebrocorticalnekrose, CCN, „Hirnrindennekrose“) ist eine bei Wiederkäuern auftretende metabolisch-toxische Gehirnerkrankung (Enzephalopathie) infolge eines Mangels an Thiamin (Vitamin B1). Sie entspricht damit der Beriberi bzw. der Wernicke-Enzephalopathie des Menschen, der Chastek-Paralyse der Pelztiere und der Thiaminmangel-Enzephalopathie der Katze.
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Polioencephalomalacia (PEM), also referred to as cerebrocortical necrosis (CCN), is a neurological disease seen in ruminants that is caused by multiple factors, one of which is thiamine depletion in the body. Thiamine (vitamin B1) is a key chemical in glucose metabolism that, when deficient, is most threatening to neurological activity. In addition to altered thiamine status, an association with high sulfur intake has been observed as a potential cause of PEM. Other toxic or metabolic diseases (eg, acute lead poisoning, sodium toxicosis/water deprivation) can result in PEM as well. Cattle, sheep, goat, and other ruminants that are diagnosed with PEM or pre-PEM suffer opisthotonus, cortical blindness, disoriented movement, and eventually fatality, if left untreated. Current data shows that the onset of PEM can range from birth to late adulthood.
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6951
