Ali Shilatifard

http://dbpedia.org/resource/Ali_Shilatifard an entity of type: Thing

Ali Shilatifard is an American biochemist/molecular biologist, the Robert Francis Furchgott Professor and Chairman of the department of biochemistry and molecular genetics, and the director of the Simpson Query Institute for Epigenetics at the Northwestern University Feinberg School of Medicine. He has served as a senior editor for the journal Science, and currently serves as the Editor for Science's open access journal Science Advances. Research in Shilatifard's lab focuses on the cause of childhood leukemia through chromosomal translocations, the role of ELL in this process, and the discovery of the Super Elongation Complex as being a central complex linking MLL translocations into a diverse number of genes to leukemic pathogenesis. rdf:langString
rdf:langString Ali Shilatifard
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rdf:langString
rdf:langString St. Louis University School of Medicine
rdf:langString Stowers Institute for Medical Research,
rdf:langString Northwestern University Feinberg School of Medicine
rdf:langString Kennesaw State University
rdf:langString
rdf:langString University of Georgia, Athens
rdf:langString Elected Fellow of the American Association for the Advancement of Science ASBMB-AMGEN Award
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rdf:langString Laura Shilatifard
rdf:langString Ali Shilatifard is an American biochemist/molecular biologist, the Robert Francis Furchgott Professor and Chairman of the department of biochemistry and molecular genetics, and the director of the Simpson Query Institute for Epigenetics at the Northwestern University Feinberg School of Medicine. He has served as a senior editor for the journal Science, and currently serves as the Editor for Science's open access journal Science Advances. Research in Shilatifard's lab focuses on the cause of childhood leukemia through chromosomal translocations, the role of ELL in this process, and the discovery of the Super Elongation Complex as being a central complex linking MLL translocations into a diverse number of genes to leukemic pathogenesis.
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